Carcinogenesis can be modulated by altering the physiological properties of the target parenchyma. Toxic cell injury, dietary, metabolic, or hormone imbalances may modify the course of chemical carcinogenesis, ultimately by altering the homeostasis of cell proliferation between the parenchymal cells and the preneoplastic lesions. The papers in this proceedings volume are divided into four sections: oncogenes and cancer development, genetic factors, metabolic regulation in cancer development, and cell death and multistage chemical carcinogenesis. Annotation copyright Book News, Inc. Portland, Or.
